Models of Aetiology of Addiction
· Disease model: emphasises the biological and genetic basis of addition. Loss of control is a central feature
·
Self-medication hypothesis: use
specific pharmacological effects to self medicate for psychological disturbance
and painful affects
· Biopsychosocial model: Multifactorial causality, interaction of genetic predisposition, biological factors, and psychological and sociocultural factors
·
Biological model: impact on mesolimbic
reward system – extends from the ventral tegmentum to the nucleus accumbens,
with projections to areas such as the limbic system and the orbitofrontal
cortex
·
Early childhood learning:
modelling by parents and significant others
·
Current environment:
reinforcement or punishment for different practices (especially if immediate)
·
Views/knowledge/beliefs about
risk: usually over-rate our health and under-rate the risks
·
Our resources (usually an excuse)
·
Physical environment (work/home)
·
Social influences (friends/media)
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