HYPOTHERMIA
Hypothermia is a condition in which the core (internal)
temper-ature is 35°C
(95°F)
or less as a result of exposure to cold. Hy-pothermia occurs when a patient
loses the ability to maintain body temperature. Urban hypothermia (extreme
exposure to cold in an urban setting) is associated with a high mortality rate;
elderly people, infants, people with concurrent illnesses, and the homeless are
particularly susceptible. Alcohol ingestion increases susceptibility because it
causes systemic vasodilation. Trauma vic-tims are also at risk for hypothermia
resulting from treatment with cold fluids, unwarmed oxygen, and exposure during
exam-ination. The patient may also have frostbite, but the hypothermia takes
precedence in treatment.
Hypothermia leads to
physiologic changes in all organ systems. There is progressive deterioration,
with apathy, poor judgment, ataxia, dysarthria, drowsiness, pulmonary edema,
acid-base ab-normalities, coagulopathy, and eventual coma. Shivering may be
suppressed below a temperature of 32.2°C
(90°F), because the body’s self-warming mechanisms
become ineffective. The heart-beat and blood pressure may be so weak that
peripheral pulses become undetectable. Cardiac irregularities may also occur.
Other physiologic abnormalities include hypoxemia and acidosis.
Management consists of continuous monitoring, rewarming,
removal of wet clothing, insulation, and supportive care.
The ABCs of basic life support are a priority. The
patient’s vital signs, CVP, urine output, arterial blood gas levels, blood
chem-istry determinations (blood urea nitrogen, creatinine, glucose,
electrolytes), and chest x-rays are evaluated frequently. Body temperature is
monitored with an esophageal, bladder, or rectal thermistor. Continuous ECG
monitoring is performed, because cold-induced myocardial irritability leads to
conduction distur-bances, especially ventricular fibrillation. An arterial line
is in-serted and maintained to record blood pressure and to facilitate blood
sampling.
Rewarming methods
include active core (internal) rewarming, active external rewarming, and
passive or spontaneous rewarming.
Core rewarming methods
include cardiopulmonary bypass,warm fluid administration, warm humidified
oxygen by venti-lator, and warmed peritoneal lavage. Core rewarming is
recom-mended for severe hypothermia. Monitoring for ventricular fibrillation as
the patient passes through 31° to 32°C (88° to 90°F) is essential.
Passive external rewarming includes the use of warm blankets orover-the-bed heaters.
Passive rewarming of the extremities increases blood flow to the acidotic,
anaerobic extremities. The cold blood with high lactic acid levels returning to
the core has significant effects on the core temperature and metabolic
response, possibly causing cardiac dysrhythmias and electrolyte disturbances.
Supportive care during rewarming includes the following
as directed:
·
External cardiac compression
(only as directed in the very cold patient)
·
Defibrillation of ventricular
fibrillation. Patients whose temperature is less than 32°C (90°F) will experience
spon-taneous ventricular fibrillation if moved or touched. Defib-rillation is
ineffective in patients with temperatures lower than 31°C (88°F).
·
Mechanical ventilation with
positive end-expiratory pres-sure (PEEP) and heated humidified oxygen to
maintain tissue oxygenation
·
Administration of warmed
intravenous fluids to correct hy-potension and maintain urine output and core
rewarming, as described previously
·
Administration of sodium
bicarbonate to correct metabolic acidosis if necessary
·
Administration of
antiarrhythmic medications
·
Insertion of an indwelling
urinary catheter to monitor fluid status
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