INTESTINAL MALROTATION & VOLVULUS
Malrotation of the intestines is a developmental abnormality that
permits spontaneous abnormal rotation of the midgut around the mesentery
(supe-rior mesenteric artery). The incidence of malrota-tion is estimated to be
about 1:500 live births. Most patients with malrotation of the midgut present
during infancy with symptoms of bowel obstruc-tion. Coiling of the duodenum
with the ascending colon can produce complete or partial duodenal obstruction.
The most serious complication of mal-rotation, a midgut volvulus, can rapidly
compro-mise intestinal blood supply causing infarction. Midgut volvulus is a
true surgical emergency that most commonly occurs in infancy, with up to one
third occurring in the first week of life. The mor-tality rate is high (up to
25%). Typical symptoms are bilious vomiting, progressive abdominal dis-tention
and tenderness, metabolic acidosis, and hemodynamic instability. Bloody
diarrhea may be indicative of bowel infarction. Abdominal ultraso-nography or
upper gastrointestinal imaging con-firms the diagnosis.
Surgery provides the only definitive treatment of malrotation and midgut
volvulus. If obstruction is present but obvious volvulus has not yet occurred,
preoperative preparation may include stabilization of any coexisting
conditions, insertion of a nasogas-tric (or orogastric tube) to decompress the
stomach, broad-spectrum antibiotics, fluid and electrolyte replacement, and
prompt transport to the operating room.
These patients are at increased risk for
pul-monary aspiration. Depending on the size of the patient, rapid sequence
induction (or awake intuba-tion) should be employed. Patients with volvulus are
usually hypovolemic and acidotic, and may tolerate anesthesia poorly. Ketamine
may be the preferred anesthetic induction agent. An opioid-based anes-thetic
can also be used as postoperative ventilation will often be necessary. Fluid
resuscitation, likely including blood products, and sodium bicarbonate therapy
are usually necessary. Arterial and central venous lines are helpful. Surgical
treatment includes reducing the volvulus, freeing the obstruction, wid-ening
the base of mesenteric attachments, and resect-ing any obviously necrotic
bowel. Bowel edema can complicate abdominal closure and has the potential to
produce an abdominal compartment syndrome. The latter can impair ventilation,
hinder venous return, and produce renal compromise; delayed fascial closure or temporary closure with a
Silastic “silo” may be necessary. A second-look laparotomy may be required
24–48 h later to ensure viability of the remaining bowel.
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