Pulmonary problems during
anesthesia
Once the
airway is secured, many things can still go wrong with the pulmonary system.
Often – but not invariably – pulse oximetry gives the first signal of trouble:
(i) We have to provide oxygen into the airway.
Problems arise when (inadver-tently) another gas is substituted for oxygen, or
when a mechanical problem affects the delivery mechanism (like failure to turn
on the ventilator or a disconnection of the ventilator from the breathing
circuit).
(ii) We need to have adequate alveolar ventilation,
i.e., tidal volumes in excess of deadspace. Problems include a kinked or
plugged ETT, a leak somewhere (allowing the gas to vent to the atmosphere),
bronchospasm, pneumothorax, a plug (mucus, blood, tissue, foreign body) in a
bronchus, decreased lung compliance, increased intrathoracic pressure (as with
insufflation of carbon dioxide into the abdomen for laparoscopy), inadequate fresh
gas flow rate, or increased apparatus deadspace as from machine valve failure.
(iii) When the oxygen arrives at the alveolus, it has
to be able to get into the blood stream. Problems here include a diffusion
block in the alveolus (pulmonary edema fluid), lack of blood flow to the
alveolus (pulmonary embolism), or inability of the blood to pick up oxygen,
e.g., carbon monoxide poisoning – though this would fool the SpO2
into reporting normal saturation; see Mon-itoring chapter).
(iv) Finally, the oxygenated blood has to make it to
the location of the pulse oxi-meter for analysis. Problems here would include
dilution of the oxygenated blood with venous blood (shunt), flow blockade to
the location of the pulse oximeter (distal to an inflated blood pressure cuff or
tourniquet), presence of dyes that can
alter the color of the blood (methylene blue), inaccurate probe placement (only
partially on the finger), or failure of the oximeter probe itself.
So, in
addition to calling for help . . .
(i) Check FiO2 (if unexpectedly low,
disconnect from wall oxygen source and use oxygen from a cylinder or room air).
(ii) Increase FiO2, e.g., turn off
nitrous oxide, increase fresh gas flow with oxygen.
(iii) Check capnogram shape of ETCO2
waveform – in short, confirm adequate gas exchange.
(iv) Check pulse oximeter waveform and probe
(reposition as needed).
(v) Listen to breath sounds bilaterally – mainstem
intubation? Pneumothorax? Inadvertent extubation?
(vi) Check peak inspiratory pressure – if low, there
may be a leak; if high, an obstruction.
(A) Give
several manual breaths – while it turns out that even “educated hands” cannot
gauge compliance and resistance well, a few slow, deep manual breaths allow
control over the pattern of inspiration, which may improve the situation.
However, we must be careful not to get stuck just squeezing the bag to feel as
if we are doing something, tying up our hands when we could be handling other
needs. Also, anesthesia machines and ventilators will generate peak inspiratory
pressure and tidal vol-ume data
(rather than impressions). Observing these two parameters during anesthesia
helps us detect trends that might herald problems before they become
emergencies.
(B ) Suction ETT – confirms patency and removes secretions.
(vii) Check exhaled tidal volume (to ensure there is no
leak).
(viii) Consider obtaining arterial blood gases and
chest radiograph.
(ix) PEEP – administering PEEP may improve the
saturation, since often the cause is decreased FRC. Inspiratory pressures
and/or venous return can constrain the level of PEEP.
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