Respiratory distress: management
Assess the patient for the
following:
·
Colour: pallor or cyanosis.
·
Respiratory drive: pattern and timing of breathing
may reflect a central or brainstem
cause.
·
Inspiration and expiration of air
at the mouth and nose: upper
airway obstruction produces stridor;
lower airway obstruction leads to cough, wheeze, and a prolonged expiratory
phase.
·
Chest wall movement: chest and abdominal wall dynamics
may indicate flail-chest,
diaphragmatic palsy, pneumothorax, or foreign body inhalation.
·
Position and level of agitation.
·
Mental state.
·
Heart rate and perfusion: these may reflect impending
arrest.
·
Non-invasive: pulse oximetry measurement of
oxyhaemoglobin saturation, pulse
oximetry measurement of oxyhaemoglobin saturation (SpO2).
·
Arterial blood gas: assessment of acid–base, PaO2,
PaCO2. A capillary blood
sample is a good alternative (for pH, PCO2) if the extremity is warm
and the blood flows freely.
·
Blood tests: full blood count (FBC),
electrolytes, glucose, and cultures.
·
CXR: for diagnosis (e.g. severe
pneumonia); for assessment of complications
(e.g. pulmonary oedema, pneumothorax).
·
Pulse
oximetry.
· Continuous ECG.
· BP.
· Temperature.
· Fluid balance.
· Conscious level.
There are specific therapies for
each condition listed in the ‘Differential diagnoses’. With regard to fluid
ther-apy, we generally restrict total volume to 80% maintenance for the
fol-lowing reasons.
·
Distress with retraction: high –ve intrathoracic pressure
will pull fluid out of capillaries
into the interstitial space and will aggravate breathing with pulmonary oedema.
·
Syndrome of inappropriate
antidiuretic hormone (SIADH): this is a common
problem in moderate to severe respiratory distress.
·
Diuresis is limited: in the hydrated patient consider
using furosemide (0.5–1mg/kg, IV). It
may help the patient with extra-interstitial water without overt oedema.
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