Abruptio Placenta
Bleeding
is due to premature separation of a normally situated placenta occurring after
22wks of pregnancy. (Sometime refered as Abruption – tear asunder) Accidental
ble eding, it is about 2% of all pregnancies. It may occur at any stage of
pregnancy or during labour.
The
aetiology of haemorrhage is not always clear (40%) but it is often associated
with hypertensive disease in pregnancy, sudden reduction in uterine size
polyhydramnious, preterm labour, rupture of membrane, acute emotional state
previous history of placenta abruption. Strenuous physical exercises over
distension of the uterus, road traffic accident, direct trauma to abdomen or
version.
Multiparty,
cigarette smoking, Poor nutritional status, Infection, after the delivery of 1st
twin – Recurs in 10-25% of cases.
In this
case blood escapes from the vagina it is the commonest type. Bleeding may
become severe from slight. It may be accompanied by abdominal pain and
tenderness, delivery of the baby should be accomplished within a few hours (6
hrs) to avoid coagulation failure developing. Bleeding is proportional to the
amount of visible vaginal blood loss.
This
hemorrhage is primarily concealed then later becomes revealed with little
vaginal bleeding. A degree of shock is exhibited which is usually severe
compared with the vaginal bleeding. It is usually associated with blood
coagulation disorders.
This is a
serious condition with high maternal and fetal mortality. It account for 55% of
maternal death. It is associated with severe bleeding but no vaginal bleeding
occurs but large retro-placental clot forms behind the placenta – maternal
surface.
Mother
shows signs and symptoms of hypovolaemic shock, uterine enlargement and severe
pain.
History
of pregnancy induced hypertension, headache, nausea and vomiting, epigastric
pain, following road traffic accident or trauma.
Mild -
general condition is fair, pain on one side of the uterus, there may or may be
no vaginal bleeding, vital signs may be normal and B/P may be raised.
Shock –
B/P may be below 90/60 or more, Severe abdo minal pain, anxiety, Uterus is
tender to touch, No fetal heart beat is heard or not heard, Pulse is rapid and
thready, signs of pre-eclampsia pitied oedema, Urine is scanty – protein is
positive or ne gative, Fetal lie is normal. Amount of visible bleeding is not a
guide to severity of the hemorrhage.
Mild
Admit,
treat as undiagnosed APH, Set up I.V. infusion Destrose 5%, Set up sintocynon
to induce labour, ARM is done if she is over 37 weeks, Observations of vital
signs, blood loss, pallor oedema and record, Vaginal delivery is contemplated.
Severe
1.
Routine blood investigation; Plasma fibrinogen and
clotting time test. The Fi-test Baxter hyland for hypofinogenaemia when
facilities for laboratory test are not available.
2.
Pethidine 100mg, morphine 15mg to relief pain
3.
Blood transfusion of at least 2 litres of fresh
blood within 1hr
rapidly .Fibrinogen 4-6g intravenously followed b y 1gm at ½ hourly
until clotting mechanism is normal – fresh blood is the best source.
4.
Monitor Renal secretion – at least 30mls per hour.
Record fluid intake – urinalysis for protein.
5.
When Clotting defect is controlled Caesarean
Section is done.
6.
Record weight of retro placental clot.
7.
EUA is done in the theatre. Rupture the membranes
to reduce intra uterine pressure and induce labour. Oxytocin is set up to start
uterine contraction.
8.
Vigilant observation of the vital signs,
contractions and Fetal heart beats.
9.
Usually labour is rapid.
10. To
prevent PPH deliver the placenta by controlled cord traction.
11. Examine
the placenta for retro placental clots .
12. Prepare
for resuscitation of an asphyxiated baby – pediatrician should be around.
1.
Observe carefully for renal function – Acute
renal failure.
2.
Restrict fluid intake to 1000mls daily.
3.
Low Protein diet, low sodium and potassium,
Estimate blood area, Potassium for 3 days.
4.
Accurate fluid balanced chart.
5.
Report signs of Oliguria (less than 500mls daily.
6.
Treat anaemia – Transfusion or give haematinics.
1.
Disseminated intravascular coagulation (DIC) – mode
rate & severe
2.
PPH. Due to convelaire uterus or DIC.
3.
Renal failure – hypovolaemia, poor kidney perfusion
.
4.
Pituitary necrosis resulting from prolonged and
severe hypotension – shock.
5.
Increased mortality for the infants – 50-80%
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