Etiology
By
definition, the ADs are stress-related phenomena in which a psychosocial
stressor results in the development of maladaptive states and psychiatric
symptoms. The condition is presumed to be time limited, that is, a transitory
reaction; symptoms recede when the stressor is removed or a new state of
adaptation is defined.
That the
relationship between stress and the occurrence of a psychiatric disorder is
both complex and uncertain has caused many to question the theoretical basis of
AD. The linear model of stress–disease interaction, which serves as the model
for AD, has been questioned as well. The linear model presupposes that direct
and clearly identifiable pathological reaction may follow a stressful event, a
scenario that no doubt occurs in some indi-viduals with AD but may not
accurately characterize others. For example, there may be multiple stressors,
insidious or chronic, as opposed to discrete events. Furthermore, relatively
minor pre-cipitating events may generate a disturbance in an individual who has
previously been sensitized to stress.
Several
authors have criticized the stressor criterion in AD because stressors are
difficult to specify and measure, and their clinical implications and impact
are uncertain. Questions pertain to whether patients with AD are unusually
sensitive to psycho-social events not likely to cause disturbance in others.
Are there individuals who have been exposed to high levels of stress, the
severity or accumulation of which would probably produce nega-tive consequences
in most people?
Diverse
variables and modifiers are involved in the pre-sentation of AD after exposure
to a stress. Cohen (1981) argued that acute stresses are different from chronic
ones in both psy-chological and physiological terms; that the meaning of the
stress is affected by “modifiers” – ego strengths, support systems, prior
mastery – and that one must differentiate manifest and latent meaning of the
stressors (e.g., loss of a job may be a relief or a catastrophe). An
objectively overwhelming stress could have little impact on one individual,
whereas another individual could regard a minor one as cataclysmic. A recent
minor stress super-imposed on a previous underlying (major) stress (which had
no observable effect on its own) may have a significant impact, not operating
independently but by its additive effect: the concatena-tion of events
(Hamburg, personal communication).
Despite
variable findings suggesting a correlation between the acknowledgment of stress
and the assignment of an AD di-agnosis, stress is not universally related to
the development of psychiatric illness, and this has implications for
understanding the meaning of stressors in AD. Specific types of stressful
events and individual patterns of stress response appear to be prefer-entially
related to the development of psychiatric symptoms in vulnerable individuals.
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