VAGINITIS AND
VAGINAL DISCHARGE
Symptomatic vaginal discharge may occur alone or
accompany salpingitis, endometritis, and cervicitis. Evaluation includes pelvic
examination, cervical cultures for N.
gonor-rhoeae and C. trachomatis, and
microscopic examination of the discharge. Measurementof the pH of the discharge
may also be helpful. Pelvic examination is valuable in deter-mining whether
uterine, adnexal, or cervical tenderness is present and whether the source of
the discharge is the cervix or the vagina.
The clinical and laboratory findings vary with the
etiologic agent. Candida albicans
generally produces a vulvovaginitis associated with pruritus and erythema of
the vulvar area and a discharge with the consistency of cottage cheese.
Microscopic demonstration of yeast and pseudomycelia in a potassium hydroxide
or Gram stain preparation of the exudate confirms the diagnosis. Trichomonas vaginalis typically produces
a foamy, puru-lent vaginal discharge. The pH is variable (usually >5.0), and
numerous polymorphonu-clear cells and motile trichomonads are seen on wet mount
examination.
Bacterial vaginosis (BV), previously termed
“nonspecific vaginitis,” is the most com-mon form of vaginitis in women. BV is
associated with overgrowth of multiple members of the vaginal anaerobic flora,
genital mycoplasmas, and a small Gram-negative rod (Gardnerella vaginalis), once believed to be the sole cause of the
disease. The vaginal discharge of BV is yellowish, homogenous, and adherent to
the vaginal wall. The pH is greater than 5.0. Addition of KOH to the vaginal
secretions produces a fishy smell as a result of volatilization of amines. The
Gram stain shows a shift from the usual lactobacil-lary flora to one of many
Gram-negative coccobacilli. Clue cells, which are vaginal epithelial cells
heavily coated with G. vaginalis, may
also be seen. Therapy depends on the etiologic agent.
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